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Perhaps less known is that female cells randomly and permanently shut off one of the X chromosomes during embryonic development through a process called X chromosome inactivation, or XCI.New research performed on mouse female embryonic stem cells by scientists at the University of California, Riverside, traces the origin of XCI to an RNA splicing mechanism.Occurring in every human cell and almost all human genes, RNA splicing copies and pastes genetic fragments scattered among the genome to create a meaningful or functional genetic message.In the case of XCI, the researchers found that a special splicing event occurs prior to XCI and in association with the X chromosome chosen for inactivation, but not with the other X chromosome.This special splicing event happens in "Xist," a gene expressed only in females and the inactive X chromosome.Upon differentiation of embryonic stem cells, Xist becomes spliced, and thus functional to kick-start subsequent events to induce XCI.
Developed by researchers at the University of Turku in Finland, an immunotherapeutic antibody therapy re-educates macrophages to activate passivated cytotoxic T cells to kill cancer.The antibody therapy prevented the growth of tumours in several mouse models.One reason behind many unsuccessful cancer treatments is the cancers' ability to hijack the immune system to support its own growth.This is assisted by the so-called tumour-associated macrophages that can be educated by cancer cells to dampen anti-tumour immune responses.Macrophages are phagocytes that form the first line of defence towards invading pathogens and they have a crucial role in maintaining tissue homeostasis.In collaboration with Academician of Science and Professor of Immunology Sirpa Jalkanen, Academy Research Fellow Maija Hollmén's research group investigated the possibility to utilise tumour-associated macrophages to increase the immunological detection and killing of cancer cells.
Ad fraud continues to be a challenge for advertisers as scammers get more sophisticated with their tactics.Fraud in digital advertising still runs rampant, particularly in mobile apps.The tactic has been around for years in websites and more recently in mobile apps and connected TVs, though the mobile app-fraud operation differs in a couple of ways.Typically, fraudsters target less popular apps that consumers are tricked into downloading.All told, Oracle estimates that the scheme that it dubbed "DrainerBot" has affected more than 10 million devices, making it one of the larger fraud operations in recent years.Read more: Mobile ad fraud continues to surge as scammers get smarter -- with in-app fraud increasing by as much as 800% this year
More than a decade ago, Logitech delighted gamers with the MX518, a high-precision mouse tooled for players who wanted performance without all the bells and whistles – and a modest price point.It’s now reviving the classic with a couple of upgrades that should make it worth a look for today’s gaming enthusiasts.The wired body of the G Series MX518 is largely unchanged from the original design: it’s got the same comfortable shape, eight programmable buttons, and glossy finish on the top panel.That seemed to be good enough for more than 15 million folks who bought the MX518 through its lifetime, starting in 2005.But this time around, it’s got Logitech‘s Hero 16K sensor, that allows for a range of up to 16,000 DPI.It’s the same one that’s on the G502 Hero that I’m currently using, and I can attest to its accuracy in both gaming and graphics applications.
One is a Logitech MX518 attached to my desktop.The other is a Logitech MX518 attached to my laptop.I refuse to purchase any other mouse and get a little anxious when I think about the mission I will have to embark on to eventually find a replacement MX518.Logitech today announced it is bringing back the “legendary” (the company’s word, not mine) MX518 gaming mouse.The announcement says “many consider [it] to be the finest gaming mouse of all time.” I am definitely one of those people.Logitech first released the MX518 in 2005, which succeeded the already-pretty-good MX510 gaming mouse released in 2004.
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When the body's immune system has just begun attacking cells of a transplanted organ, the new method's particles send a fluorescent signal into the urine.In a new study, researchers at the Georgia Institute of Technology and Emory University validated the method in a mouse model, and they have engineered the sensor with highly biocompatible components, which could make the path to potential future trials easier.The sensor particle, a nanoparticle, detects a T cell weapon, an enzyme called granzyme B, that pushes a transplanted organ's cells into the self-destruction process called apoptosis."Before any organ damage can happen, T cells have to produce granzyme B, which is why this is an early detection method," said Gabe Kwong, a co-principal investigator in the study and an assistant professor in the Wallace H. Coulter Department of Biomedical Engineering at Georgia Tech and Emory University.Kwong and Adams published the study's results in the journal Nature Biomedical Engineering on February 18, 2019.It is double-coated with dextran, a sugar, and polyethylene glycol, a common ingredient in laxatives, to keep the body from disposing of it too quickly.
Wireless mice in 2019 are nothing to sneeze at.Logitech, Steelseries and other players have been able to make mice that are completely input lag free and have batteries don't add a ton of weight to the mouse.After hours of testing, recharging and gaming with dozens of wireless gaming mice, we think that the Logitech G Pro Wireless is by-far the best wireless gaming mouse out right now.Its combination of light weight, flawless sensor and great battery life make it an obvious choice, if you can afford it.To find out the rest of our top 5 wireless choices, please read the full article here: 
DURHAM, N.C. -- Researchers at Duke University have shown that a single systemic treatment using CRISPR genome editing technology can safely and stably correct a genetic disease -- Duchenne muscular dystrophy (DMD) -- for more than a year in mice, despite observed immune responses and alternative gene editing outcomes.In 2016, Charles Gersbach, the Rooney Family Associate Professor of Biomedical Engineering at Duke, published one of the first successful uses of CRISPR to treat an animal model of genetic disease with a strategy that has the potential to be translated to human therapy.Gersbach's latest research focuses on a mouse model of DMD, which is caused by the body's inability to produce dystrophin, a long protein chain that binds the interior of a muscle fiber to its surrounding support structure.His lab was one of the first to begin focusing on CRISPR/Cas9, a modified version of a bacterial defense system that targets and slices apart the DNA of invading viruses.His approach uses CRISPR/Cas9 to snip out dystrophin exons around the genetic mutation, leaving the body's natural DNA repair system to stitch the remaining gene back together to create a shortened -- but functional -- version of the dystrophin gene."It is widely believed that gene editing leads to permanent gene correction," Gersbach said.
Pairs of swing-dancing electrons do the Lindy Hop in "Superconductivity: The Musical," the winning video for this year's geektastic Dance Your PhD contest.Pramodh Yapa, a graduate student at the University of Alberta, Canada, beat out roughly 50 other entries for the interpretive dance based on his master's thesis, "Non-Local Electrodynamics of Superconducting Wires: Implications for Flux Noise and Inductance."The Dance Your PhD contest was established in 2008 by science journalist John Bohannon, and is sponsored by Science magazine and the American Association for the Advancement of Science (AAAS).So he put together a dance party at Austria's Institute of Molecular Biotechnology, including a contest for whichever candidate could best explain their thesis topics with interpretive dance.There are four broad categories: physics, chemistry, biology, and social science, with a fairly liberal interpretation of what topics fall under each.Over the years, the quality of the videos has improved a bit—Bohannon recalled the first year's winning video just had a postdoc chasing after a couple of graduates to demonstrate mouse genetics—as has the prizes offered.
The Insider Picks team writes about stuff we think you'll like.Presidents' Day Weekend is the first time to catch a bunch of great deals after the holidays, especially for tech.We've broken them down into six categories: computers, smart home accessories, headphones and speakers, TV and home theater, smartwatches and fitness bands, vacuums, and Apple products.There are tons of deals happening at many stores, so if you're shopping for new tech, but don't know where to start, we've combed through sales at Best Buy, Walmart, Dell, and Lenovo to find 70+ deals that are worth your time.There are a handful of exceptions, and we've called them out for your convenience.Tech deals come around often, but Presidents' Day weekend gives you the opportunity to save on the all-new MacBook Air and LG's premium OLED TVs, which don't go on sale often.
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An unopened copy of Super Mario Bros. for the Nintendo Entertainment System (NES) sold for more than $100,000.The cartridge was an 1985 sticker-sealed reprint that was only available in the New York and LA to test-launch the NES.The game was sold through Heritage Auctions on Feb. 6 for $100,105 to a group of collectors.The game's box has a near-mint rating of 9.4 by Wata Games a website dedicated to rating collectible video games.The sticker has a seal rating of A++, which adds a great deal of value to the game."In terms of rarity, popularity, and relevance to collectors, this game has it all," said Wata Games President Deniz Kahn.
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Twitch, the popular video gaming streaming service, suspended the account of popular DJ deadmau5 (pronounced "dead mouse") for using homophobic slurs in an outburst while streaming "PlayerUnknown's Battlegrounds."Popular recording artist deadmau5 has been suspended from Twitch, a popular video game streaming service, for using homophobic slurs on his channel.During his evening stream of "Playerunknown's Battlegrounds" on February 10th, deadmau5 launched into a diatribe against a player who had killed him, calling him a "f****** c***-s**** stream sniper f**."As clips of the outburst began to circulate, Twitch responded by suspending deadmau5's account for hate speech.Deadmau5, who's real name is Joel Zimmerman, apparently responded to the suspension in a February 12th Reddit post that has since been deleted.The DJ began by accusing Twitch of censoring him and holding a double standard, and said he would no longer partner with the company.
When we interact with computers today we move the mouse, we scroll the trackpad, we tap the screen, but there is so much that the machines don’t pick up on — what about where we’re looking, the subtle gestures we make and what we’re thinking?Asteroid is looking to get developers comfortable with the idea that future interfaces are going to take in much more biosensory data.The team has built a node-based human-machine interface engine for macOS and iOS that allows developers to build interactions that can be imported into Swift applications.“What’s interesting about emerging human-machine interface tech is the hope that the user may be able to ‘upload’ as much as they can ‘download’ today,”Asteroid founder Saku Panditharatne wrote in a Medium post.To bring attention to their development environment, they’ve launched a crowdfunding campaign that gives a decent snapshot of the depth of experiences that can be enabled by today’s commercially available biosensors.Asteroid definitely doesn’t want to be a hardware startup, but their campaign is largely serving as a way to expose developers to what tools could be in their interaction design arsenal.
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These days, across the British Isles, borrowing cats from friends and family is apparently a thing.Cat borrowers get to enjoy kitty cuddling and mouse-murdering for free, while the wee beasties’ actual owners go broke paying vet bills.We meet several such tabby takers in a series of silly spots created by adam in London for Dreamies cat treats, a Mars Petcare brand.In each case, non-cat-owning humans entice felines into hanging out with them, using Dreamies as bait.First up, a pair of pint-sized purrr-loiners with an Addams Family vibe:Maybe it’s time to borrow a child psychologist for those two creepy kids.
CAMBRIDGE, MA -- DNA-repair enzymes help cells survive damage to their genomes, which arises as a normal byproduct of cell activity and can also be caused by environmental toxins.However, in certain situations, DNA repair can become harmful to cells, provoking an inflammatory response that produces severe tissue damage.MIT Professor Leona Samson has now determined that inflammation is a key component of the way this damage occurs in photoreceptor cells in the retinas of mice.About 10 years ago, she and her colleagues discovered that overactive initiation of DNA-repair systems can lead to retinal damage and blindness in mice.Aag glycosylase helps to repair DNA damage caused by a class of drugs known as alkylating agents, which are commonly used as chemotherapy drugs and are also found in pollutants such as tobacco smoke and fuel exhaust.Retinal damage from these drugs has not been seen in human patients, but alkylating agents may produce similar damage in other human tissues, Samson says.