Summary - A new market study, titled “Tardive Dyskinesia Market Report: Focus on US & Japan (2016-2025) ” has been featured on WiseGuyReports.
Tardive dyskinesia is an involuntary movement disorder that typically arises from the long-term use of antipsychotic medication in schizoaffective disorders. Tardive dyskinesia is characterized by involuntary, repetitive movements of the face, trunk, or extremities, including lip smacking, grimacing, tongue protrusion, facial movements or blinking, puckering and pursing of the lips. TDs are most common in patients with schizophrenia, schizoaffective disorder, or bipolar disorder who have been treated with antipsychotic medication for long periods, but they occasionally occur in other patients as well
The key factors driving the growth of the Tardive dyskinesia market are rising schizophrenia patients, increasing bipolar patients, growing antipsychotic prescription, increased use of long-term neuroleptic drugs and increasing incidence of neurological disorders. However, the expansion of the market is hindered by side effects of drugs and under-diagnosed.
The report “Tardive Dyskinesia Market” provides a comprehensive study of the US and Japan tardive dyskinesia market with detailed analysis of market size, growth and penetration of the industry. The major trends, growth drivers as well as issues being faced by the industry are being presented in this report. The industry comprises few large players such as Valeant Pharmaceuticals, Neurocrine Biosciences and Teva Pharmaceuticals Industries Ltd. All these companies have been profiled in the present report highlighting their key financials and business strategies for growth
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Tardive dyskinesia (TD) is a disorder caused by a prolonged use of antipsychotic drugs by patients suffering from schizophrenia and other neuroleptic disorders.
Although tardive dyskinesia commonly occurs in patients who have been treated with antipsychotic medication for long periods, it also occurs occasionally in other conditions such as fetal alcohol syndrome, developmental disabilities, and other brain disorders, even after receiving 1 dose of causative agent.
The disorder is linked with polymorphism of dopamine receptor gene types.
However, side effects of drugs, lack of awareness about the disorder and its medication, and undiagnosed cases are restricting the market growth.Request Brochure – https://www.transparencymarketresearch.com/sample/sample.php?flag=B_id=28283 The global tardive dyskinesia treatment market can be segmented based on type of disorder, drug class, and region.
In addition, a variety of medicines are used for the treatment of tardive dyskinesia such as clonidine; however, dose limiting hypotension and sedation are its restraining factors.Request COVID Analysis on Tardive Dyskinesia (TD) Treatment Market – https://www.transparencymarketresearch.com/sample/sample.php?flag=covid19_id=28283 Botox injections are tried for minor dystonia.
Vitamin B6, vitamin E, and branched chain amino acids are reported to be helpful to minimize the symptoms of the disorder.
This neurodegenerative disorder causes gradual breakdown and death of dopamine-producing neurons in a particular area of the brain, which is called substantial Ingra.
This fall in dopamine level causes abnormal brain activity, impaired movement, and other symptoms.There is no cure for Parkinson’s disease, at least as of now.
Most of the time these movements are slow, almost dance-like, but in some cases, the movements are rapid, like a sudden jerk or extended muscle spasms.
The important thing to note is that Dyskinesia is not a symptom of Parkinson’s, but a side effect of the medication.Causes of Dyskinesia:Patients, who are being treated with levodopa often show symptoms of Dyskinesia.
Younger people affected with PD, who are given levodopa, develop motor fluctuations and dyskinesia earlier than others.When a patient takes a dose of levodopa, it takes some time, before the drug starts its effect.
This phenomenon is called ‘wearing off’ (also known as ‘early wearing off).The terms ‘on’ and ‘off’ with regards to Parkinson’s refer to the switch between mobility and immobility in patients being treated with levodopa.
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